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Due to frequent vandalism. One huge rock has been called "Newspaper rock" and depicts a wealth of unusual and varied petroglyphs. Several robust plants of yellow saucers Malocothrix sochoides ; were inside the fence, but exotics, russian thistle Salsola australis ; and sand-butt Ambrosia acanthicarpa ; were numerous throughout the parking area and along the fence due to continued sheep and cattle trailing throughout the area. A portion of this exotic invasion has recently been removed by Department of Forestry crews during a cooperative Department of Fish and Game and BLM Fish Slough exotic plant removal project. Lunch was eaten against, or on the broken boulders of the Red Rock Canyon Petroglyph site. Many figures were pecked into the flat expanses of rock. Before us, across the valley, rose the flanks of the White Mountains. After lunch we followed a road that turns westerly, rising up to approach the mouth of a striking portion of Red Rock Canyon. Our caravan parked and we walked into this narrow, twisted, gouged and hollowed serpentine of nature's whimsy. Mostly in tones of red or pink, the rocks shelter plants in pockets or strips of soil: granite gilia Leptodactylon pungens ; , wishbone bush Mirabalis bigelovii ; , apricot mallow Spharalcea ambigua ; , and robust, old bushes of desert bitterbrush Purshia glandulosa ; . A dry look-alike for desert plume Stanleya pinnata ; proved to be Thelypodium lacinatum. And there were still many untrammeled annuals; Phacelia, Cryptantha, poppy and tidy-tips. The canyon was a fitting finale to a varied ethnobotanical and archaeological field trip held in perfect weather.
The purpose of this chapter is to provide an overview of the approximate geographic distribution and prevalence of echinococcosis, and the most important transmission cycles in various parts of the world. However, it is difficult to draw a clear picture of the current situation because published data are quite often incomplete period and area of study not mentioned, number of examined individuals not given, methods of data sampling and assessment not described, only percent values presented, wrong use of the terms prevalence and incidence, criteria for the description of high or low prevalence not clearly defined, etc. ; , some data are only listed in reports or in abstracts which are not generally accessible via databases, and for some countries or regions information is lacking. Therefore, an attempt was made to characterise the epidemiological situations by presenting selected examples of prevalence or incidence rates of echinococcosis which may not be representative for a given country region but can provide at least some basic information on the magnitude of the problem. For an improved world-wide epidemiological assessment, a new approach for uniform data collection is needed urgently
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Expanded to a .3 million year enterprise, an increase of more than 23% over the last year alone. Patients are much more savvy about their medical conditions and their treatment options. They often arrive at the doctor's office, literature search in hand, requesting the latest, and often the most expensive, new therapy. Approximately 85% of patients in the US are now covered by a third party payer for prescription costs and as a result, they pay a lower, flat rate co-pay ; for their medications. This is a driving factor for both cost and consumer demand. Co-pays for prescriptions are relatively low when compared to the prescription cost, and there is no incentive for either the patient or the provider to write for an equally effective, but lower cost alternative drug. While 60% of third party payers offer a tiered patient payment structure, with a higher co-pay on brand name drugs than the copay for generic drugs, the difference in cost is not sufficient to change prescribing behavior. Most patients do not pay proportionately for the total cost of the prescription, although such price restructuring has been advocated.
Thank you for taking the time to complete this application and for your interest in the position. Please forward the completed application form in an envelope marked PRIVATE AND CONFIDENTIAL to Ms Caroline Moss KATO Precision UK ; Limited, Mill Park Way, Off Station Road, Southwell, Nottinghamshire, NG25 0ET and kava.
Each brown tablet contains microcrystalline cellulose; ferrous fumarate; magnesium stearate; povidone; sodium starch glycolate; sucrose with modified dextrins.
Decreased mental function, confusion, seizures, weakness, flaccid paralysis can occur because of hypoxia, hyperkalemia, and decreased pH of CNS fluid. Protects client from injury resulting from decreased mentation convulsions. Acidemia may be manifested by changes in ECG configuration and presence of bradydysrhydythmias as well as increased ventricular irritability such as fibrillation signs of hyperkalemia ; . Life-threatening cardiovascular collapse may also occur because of vasodilation and decreased cardiac contractility. Note: Hypokalemia can occur as acidosis is corrected, resulting in premature ventricular contractions PVCs ; ventricular tachycardia. Deep, rapid respirations Kussmaul's ; may be noted as a compensatory mechanism to eliminate excess acid; however, as potassium shifts out of cell in an attempt to correct acidosis, respirations may become depressed. Transient respiratory depression may be the result of overcorrection of metabolic acidosis with sodium bicarbonate. Evaluates circulatory status, tissue perfusion, effects of hypotension. In the presence of coexisting hyperkalemia, GI distress e.g., distention, diarrhea, and colic ; may occur. Marked dehydration may be present because of vomiting, diarrhea. Therapy needs are based on underlying cause and fluid balance. Kidneys attempt to compensate for acidosis by excreting excess hydrogen in the form of weak acids and ammonia. Maximum urine acidity is pH of Neutralizes mouth acids and provides protective lubrication and kenalog.
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Cunha-Vaz J. Coimbra P ; 2709-16.05 Characterization of diabetic retinopathy phenotypes by multimode imaging Soubrane G. Crteil F ; ICG-Angiography in AMD Lewis H. Cleveland USA ; OCT in clinical practice in macular diseases 2709-16.06.
The factors that determine the risk of developing clinical CaP are not well-known; however, a few have been identified. The most important risk factor seems to be heredity - if one first-line relative brother or father ; has the disease, the risk is doubled. If two or more first-line relatives are affected the risk increases to 5-11-fold 5, 6 ; . A small subpopulation of individuals with CaP about 9% ; has true hereditary CaP, defined as three or more relatives affected or at least two who develop early-onset disease before age 55 years ; 7 and keppra.
1.3 Drug Needs Estimates Under SPC Regimes Table 1: Drug Needs Algorithm Parameter Label Descriptions N t Total population in year t PGt Year t population growth rate Sc Screening compliance rate NSt Number to participate in year t screening Pt Year t overall schistosomiasis prevalence rate MPt Year t change in prevalence rate SR Kato test sensitivity GAF t Gross adjustment factor in year t LM % of the population lactating babies AGE % of the population below 2 years A H % absent from home during treatment EM % of mothers expecting babies N C % of the population that would not comply with therapy ARt % in severe and comatose states OT % on other treatments GAF LM + AGE + AH + ARt + OT AGE1 % of the infected in aged 15 years AGE2 % of the infected in aged 15 years KG1 Average weight of those aged 15 years KG2 Average weight of those aged 15 years D P Praziquantel dose per kilogramme of body weight D O Oxamniquine dose per kilogramme of body weight M P milligramme per praziquantel tablet M O milligramme per oxamniquine capsule W % of capsules or tablets to be spoilt or wasted or lost DRUGt Quantity of drugs needed in year t NIt Number infected in year t NAGE1 t Number of those infected aged 15 years in year t NAGE2 t Number of those infected aged 15 years in year t NIDt Number of those infected to be detected in the test NIDEt Number of positives eligible for treatment NAGEKG1t Total body weight of those aged 15 years NAGEKG2t Total body weight of those aged 15 years NAGEKG t Grand total body weight in year t DRUGP t Total praziquantel drug needs in year t DRUGO t Total oxamniquine drug needs in year t ADRUGP t DRUGPt adjusted for wastage and loss in year t ADRUGO t DRUGO t adjusted for wastage and loss in year t.
Acknowledge supports from the materials and manufacturing directorate of the air force research laboratory and ketek.
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Composition of beef in Slovak ; . In: Proceedings of the Conference "Nutrition and Foods for 3rd Millennium", Nitra, Slovak Republic, April 24. 25. 2002, p. 243 249 Staruch L., Volansk P., Lederleitnerov J., Budajov J., Vrankov E., Stullerov .: Vskyt a obsah polyfosftov vo vybranch msovch vrobkoch. Content of polyphosphate in selected meat products in Slovak ; . In: Proceedings of the 33rd Symposia New Trends of Food Production and Evaluation", Skalsk Dvr, Czech Republic, May 27. - 29. 2002, p. 24 Staruch L., Volansk, P., Lederleitnerov J., Pipek P., Jelenkov J., Golian J.: Polyfosfty v msovch vrobkoch. Polyphosphate in meat products in Slovak ; . In: Proceedings of the Conference Detva, Slovak Republic, June 17.-19. 2002, p 140 Stefnikov Z., Jurkovicov J., Sevckov ., Sekretr S., ghov .: Vplyv behaviorlnych a psychosomatickch faktorov na vzivov spotrebu vybranej populcie. Effect of behavioural and psychosomatic factors on nutritional consumption of selected population in Slovak ; . In: Proceedings of the Conference "Environment and Health ", Bratislava 2002, p. 168-174 lecture ; Stefnikov Z., Jurkovicov J., Sevckov ., Sekretr S., ghov .: Vsledky longitudinlnej stdie zivotnho stlu th slovenskch vysokoskolkov. Results of longitudinal study of the student's lifestyle In: Proceedings of the 30 Conference " Days of children and youth ", Roznov pod Radhostm, Czech Republik, May 14.-16. 2002, p. 57-62 lecture ; Svaov ., Hesov J., Breierov, E., Augustn J., Stratilov E.: Effect of pH on the production of multiple forms of polygalacturonidase by there strains of Aureobasidium pullulans. In: Proceedings of the International Conference about Yeasts, Smolenice, Slovak Republic, May 29. - 31. 2002 poster ; Valk ., Grner F., Laukov D.: Hodnotenie hygienickej bezchybnosti a validcia predikcie trvanlivosti pasterizovanho mlieka z hadiska Bacillus cereus. Evaluation of hygienic character and validity of pasterized milk durability according to Bacillus cereus in Slovak ; . In.: Proceedings of papers from the Seminar "Milk and Chees", Praha, Czech Republic, January 22-23. 2002, p. 143-147 lecture and ketoprofen.
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T. F. Liischer and R. R. Wenzel T, Goto K, Furuichi Y, Koide H. Renal expression of mRNAs for endothelin-1, endothelin-3 and endothelin receptors in NZB W Fl mice. Renal Physiol Biochem 1993; 16: 233-243. Vanhoutte PM. Is endothelin involved in the pathogenesis of hypertension? Hypertension 1993; 21: 747-751. Vierhapper H, Wagner OF, Nowotny P, Waldhausl W. Effect of endothelin-1 in man: pretreatment with nifedipine, with indomethacin and with cyclosporine A. Eur J Clin Invest 1992; 22: 55-59. Yokokawa K, Tahara H, Kohno M, Murakawa K, Yasunari K, Nakagawa K, Hamada T, Otani S, Yanagisawa M, Takeda T. Hypertension associated with endothelin-secreting malignant hemangioendothelioma. Ann Intern Med 1991; 14: 213-215. Wagner OF, Christ G, Wojta J, Vierhapper H, Parzer S, Nowotny PJ, Schneider B, Waldhausl W, Binder BR. Polar secretion of endothelin-1 by cultured endothelial cells. J Biol Chem 1992; 267: 16066-16068. Suzuki N, Miyauchi T, Tomobe Y, Matsumoto H, Goto K, Masaki T, Fujino M. Plasma concentrations of endothelin-1 in spontaneously hypertensive rats and DOCA-salt hypertensive rats. Biochem Biophys Res Commun 1990; 167: 941-947. Kitamura K, Tanaka T, Kato J, Ogawa T, Eto T, Tanaka K. Immunoreactive endothelin in rat kidney inner medulla: marked decrease in spontaneously hypertensive rats. Biochem Biophys Res Commun 1989; 62: 38-44. Kurihara Y, Kurihara H, Suzuki H, Kodama T, Maemura K, Nagai R, Oda H, Kuwaki T, Cao W-H, Kamada N, Jishage K, Ouchi Y, Azuma S, Toyoda Y, Ishikawa T, Kumada M, Yazaki Y. Elevated blood pressure and craniofacial abnormalities in mice deficient in endothelin-1. Nature 1994; 368: 703-710. Ong AC, Jowett TP, Scoble JE, O'Shea JA, Varghese Z, Moorhead JF. Effect of cyclosporin A on endothelin synthesis by cultured human renal cortical epithelial cells. Nephrol Dial Transplant 1993; 8: 748-753. Bunchman TE, Brookshire CA. Cyclosporine-induced synthesis of endothelin by cultured human endothelial cells. J Clin Invest 1991; 88: 310-314. Iwasaki S, Homma T, Kon V. Site specific regulation in the kidney of endothelin and its receptor subtypes by cyclosporine. Kidney Int 1994; 45: 592-597. Copeland KR, Yatscoff RW. Comparison of the effects of cyclosporine and its metabolites on the release of prostacyclin and endothelin from mesangial cells. Transplantation 1992; 53: 640-645. Moutabarrik A, Ishibashi M, Fukunaga M, Kameoka H, Kawaguchi N, Takano Y, Kokado Y, Sonoda T, Onishi S, Takahara S, et al. FK506-induced kidney tubular cell injury. Transplantation 1992; 54: 1041-1047. Lanese DM, Conger JD. Effects of endothelin receptor antagonist on cyclosporine-induced vasoconstriction in isolated rat renal arterioles. J Clin Invest 1993; 91: 2144-2149. Grieff M, Shoheib SA, Ldrtscher R, Stewart DJ. Cyclosporine A induces elevation in circulating endothelin-1 following transplantation. J Soc Nephrol 1990; 1: 758. Kon V, Awazu M. Endothelin and cyclosporine nephrotoxicity. Renal Fail 1992; 14: 345-350.
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Zhdanova IV, Wurtman RJ, Lynch HL, Ives JR, Dollins AB, Morabito C, et al . Sleep-inducing effects of low doses of melatonin ingested in the evening. Clin Pharmacol Ther. 1995; 57 5 ; : 552-8. Zhdanova IV, Wurtman RJ, Morabito C, Piotrovska VR, Lynch HJ. Effects of low oral doses of melatonin given 2-4 hours before habitual bedtime on sleep in normal young humans. Sleep. 1996; 19 5 ; : 423-31. Nave R, Peled R, Lavie P. Melatonin improves evening napping. Eur J Pharmacol. 1995; 275 2 ; : 213-6. Attenburrow ME, Cowen PJ, Sharpley AL. Low dose melatonin improves sleep in healthy middle-aged subjects. Psychopharmacology Berl ; . 1996; 126 2 ; : 179-81. Pinto LR Jr, Seabra ML, Tufik S. Different criteria of sleep latency and the effect of melatonin on sleep consolidation. Sleep. 2004; 27 6 ; : 1089-92. Brusco LI, Fainstein I, Marquez M, Cardinali DP. Effect of melatonin in selected populations of sleep-disturbed patients. Biol Signals Recept. 1999; 8 1-2 ; : 126-31. Fainstein I, Bonetto AJ, Brusco LI, Cardinali DP. Effects of melatonin in elderly patients with sleep disturbance: a pilot study. Curr Ther Res. 1997; 58: 990-1000. Woodward M. Insomnia in the elderly. Aust Fam Physician. 1999; 28 7 ; : 653-8. Miyamoto M, Nishikawa H, Doken Y, Hirai K, Uchikawa O, Ohkawa S. The sleep-promoting action of ramelteon TAK-375 ; in freely moving cats. Sleep. 2004; 27 7 ; : 1319-25. Panzer A, Viljoen M. The validity of melatonin as oncostatic agent. J Pineal Res. 1997; 22 4 ; : 184-202. Rodriguez C, Mayo JC, Sainz RM, Antolin I, Herrera F, Martin J, et al. Regulation of antioxidant enzymes: a significant role for melatonin. J Pineal Res. 2004; 36 1 ; : 1-9. Nelson RJ, Demas GE, Klein SL, Kriegsfeld LS. The influence of season, photoperiod, and pineal melatonin on immune function. J Pineal Res. 1995; 19 4 ; : 149-65. Michaud M, Dumont M, Selmaoui B, Paquet J, Fantini ML, Montplaisir J. Circadian rhythm of restless leg syndrome: relationship with biological markers. Ann Neurol. 2004; 55 3 ; : 372-80. Cohen AS, Kraube H. Rare nocturnal headaches. Curr Opin Neurol. 2004; 17 3 ; : 295-9. Spence DW, Kayumov L, Chen A, Lowe A, Jain U, Katzman MA, et al. Acupuncture increases nocturnal melatonin secretion and reduces insomnia and anxiety: a preliminary report. J Neuropsychiatry Clin Neurosci. 2004; 16 1 ; : 19-28. Harinath K, Malhotra AS, Pal K, Prasad R, Kumar R, Kain TC, et al. Effects of Hatha yoga and Omkar meditation on cardiorespiratory performance, psychologic profile, and melatonin secretion. J Altern Complement Med. 2004; 10 2 ; : 261-8. Foster AC, Pelleymounter MA, Cullen MJ, Lewis D, Joppa M, Chen TK, et al. In vivo pharmacological characterization of indiplon, a novel pyrazolopyrimidine sedative-hypnotic. J Pharmacol Exp Ther. 2004; 311 2 ; : 547-59. Kato K, Hirai K, Nishiyama K, Uchikawa O, Fukatsu K, Ohkawa S, et al. Neurochemical properties of ramelteon TAK-375 ; , a selective MT 1 ; MT receptor agonist. Neuropharmacology. 2005; 48 2 ; : 301-10. Zammit GK, McNabb LJ, Caron J, Amato DA, Roth T. Efficacy and safety of eszopiclone across 6-weeks of treatment for primary insomnia. Curr Med Res Opin. 2004; 20 12 ; : 1979-91 and kato.
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Blood of Ascidia mentula and noted their resistance to the ordinary laboratory reagents; but that strong acids, potash, and ammonia cause all the orange corpuscles to disappear more or less rapidly. I, too, have found them to be resistant to the ordinary laboratory reagents. Sectioned material following fixation in Bouin'sfluidwithout acetic acid shows these bodies very well preserved. Azema 19296, c ; has re-examined the pigment in the orange cells of the Synoicidae and the Botryllidae, and reached the conclusion that it is a carotinoid. These cells are the rule in ascidian blood, but seem not to be universal. Cuenot noted the absence of orange cells in some species. I have not found them in Clavelina oblonga, Clavelina picta, Distaplia bermudensis, Lissoclinum fragile, Molgula m a n Aplidium Amaroucium ; stellatum, and Botrylloides nigrum. However, Azema 19296 ; reports orange cells to be present in the species of Botryllidae studied by him. It is possible that a more diligent search might reveal a few orange cells in some of the species in which I have failed tofindthem. They are much more abundant in some species than in others, and it is probable that there is considerable numerical variation among individuals of the same species. These cells are present in the circulating blood, and distributed here and there in the tissues. They are most abundant in the tissues of the mantle in the neighbourhood of the siphons, especially near their tips, where they frequently form spots and splotches. They become progressively less abundant as one passes towards the bases of the zooids. The carotinoid granules need not be limited to specific cells: they are present in the cytoplasmic septa of the green cells of Symplegma viride and Diplosoma macdonaldi; in all blood-cell types but not all blood-cells ; o f P y Polycarpa o b t and Polycitor Eudistoma ; olivaceus; and in the cells of most if not all tissues and organs of Didemnum Polysyncraton ; amethysteum and Polyandrocarpa t i n least some of the species Clavelina oblonga ; in which no orange cells have been found in the blood, orange and klonopin.
N engl j med 1991; 325: 58 masaoka t, takaku f, kato s et al: recombinant human granulocyte colony-stimulating factor in allogeneic bone marrow transplantation.
Te a m National Capital Area visited the Johns Hopkins laboratory September 9 to learn about their pancreatic cancer research programs. Dr. Ralph Hruban gave an overview of the lab's growth, then other staff gave presentations. TeamHOPE thanks the staf f at Johns Hopkins for this ver y informative tour and kytril.
The question of the safety of lactation during ATD therapy arises in several clinical situations. The activity of Graves' disease fluctuates through pregnancy, with improvement in the third trimester and subsequent reduction or discontinuation of ATD therapy, only to be followed by worsening of the disease in the postpartum period with resumption of ATD therapy. Some women with Graves' disease may require continuous ATD therapy until delivery and would like to begin nursing immediately. Others may have previously stopped ATD therapy late in pregnancy and are currently nursing, but subsequently experience an exacerbation of hyperthyroidism requiring ATD initiation; however, they wish to continue breast-feeding. In addition, Graves' disease may first occur in the postpartum period, either as the initial manifestation of hyperthyroidism or following postpartum thyroiditis 46 ; . Again, if such women are currently nursing, they often desire to continue even if ATD therapy is required. In the past, women taking ATD therapy have been advised against breast-feeding because of the presumption that the concentrations of these drugs in breast milk were sufficient to affect the infant's thyroid. This recommendation derives from studies of thiouracil, one of the first thiourea compounds used for treatment of thyrotoxicosis. In 1944, Williams et al. 47 ; reported that the thiouracil concentration in breast milk was 3 times higher than that in serum 2 h after oral administration of 1 g thiouracil to two lactating women. Although thiouracil treatment of hyperthyroidism was soon discontinued for other reasons, no studies of the currently used ATD drugs were published until 20 yr ago and kava.
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598. Toivola PTK, Gale CC, Goodner CJ 1972 Central alpha-adrenergic regulation of growth hormone and insulin. Hormones 3: 193196 599. Chihara K, Kodama H, Kaji H, Kita T, Kashio Y, Okimura Y, Abe H, Fujita T 1985 Augmentation by propranolol of growth hormone releasing hormone 1 44 ; NH2-induced growth hormone releas in normal short and normal children. J Clin Endocrinol Metab 61: 229 233 Mauras N, Blizzard RM, Thorner MO, Rogol AD 1987 Selective B1adrenergic receptor-blockade with atenolol enhances basal and growth hormone releasing hormone mediated growth hormone release in man. Metabolism 36: 369 372 Blackard W, Heidingsfelder S 1968 Adrenergic receptor control mechanism for growth hormone secretion. J Clin Invest 47: 14071414 602. Perkins SN, Evans WS, Thorner MO, Cronin MJ 1983 Beta-adrenergic stimulation of growth hormone release from perifused rat anterior pituitary cells. Neuroendocrinology 37: 473 475 Samols E, Weir GC, Ranseur R, Day JA, Patel YC 1978 Modulation of pancreatic somatostatin by adrenergic and cholinergic agonism and by hypoglycemic sulfonamides. Metabolism 9: 1219 1223 Richardson SB, Twente S 1990 Inhibition of hypothalamic somatostatin release by beta-adrenergic antagonist. Endocrinology 126: 10431046 605. Krieg RJ, Perkins SN, Johnson JH 1988 Beta-adrenergic stimulation of growth hormone GH ; release in vivo and subsequent inhibition of GH releasing factor-induced GH secretion. Endocrinology 122: 531535 606. Kelijman M, Frohman LA 1989 Beta-adrenergic modulation of growth hormone GH ; -autofeedback on sleep-associated and pharmacologically induced GH secretion. J Clin Endocrinol Metab 69: 11871193 607. Massara F, Camanni F 1972 Effect of various adrenergic receptor stimulating and blocking agents on human growth hormone secretion. J Endocrinol 54: 195206 608. Giustain A, Malerba M, Bresciani E, Desenzani P, Licini M, Zaltieri G, Grassi V 1995 Effect of two 2-agonist drugs, salbutamol and broxaterol, on the growth hormone response to exercise in adult patients wtih asthmatic bronchitis. J Endocrinol Invest 18: 847 852 Murakami Y, Kato Kabayama Y, Tojo K, Inoue T, Imura H 1986 Involvement of growth hormone releasing factor in GH secretion induced by serotoninergic mechanism in conscious rat. Endocrinology 119: 1089 1092 Cataldi M, Magnan E, Guillaume V, Hery F, Dutour A, Rettori MC, Kamoun A, Oliver C 1994 Effect of tianeptine on the hypothalamic somatotropic axis in the conscious sheep. Eur J Pharmacol 253: 149 153 Mueller EE, Udeschini G, Secchi C, Zambotti F, Vicentini L, Panterai F, Cocola F, Mantegazza P 1976 Inhibitory role of the serotoninergic system in hypoglycemia-induced growth hormone release in the dog. Acta Endocrinol Copenh ; 82: 7178 612. Imura H, Nakai I, Yoshimi T 1973 Effect of 5-hydrotryptophan on growth hormone and ACTH release in man. J Clin Endocrinol Metab 39: 15 613. Parati EA, Zanardi P, Cocci D, Caraceni T, Mueller EE 1980 Neuroendocrine effect of quipazine in man in healthy state and in neurological disorders. J Neural Transm Gen Sect 47: 273297 614. Delitala G, Devilla L, Bionda S, Franca V 1977 Suppression of human GH secretion by cyproheptadine. Metabolism 26: 931935 615. Casanueva FA, Villanueva LPA, Cabezas-Cerrato J 1984 Depending on the stimulus, central serotoninergic activation by fenfluramine blocks or does not alter growth hormone secretion in man. Neuroendocrinology 38: 302308 616. Biwens CH, Lebovitz HE, Feldman JM 1973 Inhibition of hypoglycemiainduced GH secretion by the serotonin antagonists cyproheptadine and methysergide. N Engl J Med 289: 236 238 Hoyer D, Clarke DE, Fozard JR 1994 International Union of Pharmacology classification of receptors for 5-hydroxytryptamine serotonin ; . Pharmacol Rev 46: 157203 618. Mota M, Bento A, Penalva A, Pombo M, Dieguez C 1995 Role of the serotonin receptor subtype 5-HTD in basal and stimulated growth hormone secretion. J Clin Endocrinol Metab 80: 19731977 619. Peroutka SJ, McCarthy BG 1989 Sumatriptan GR43157 ; interacts selectively with 5-HT1B and 5-HT1D binding sites. Eur J Pharmacol 163: 133136 620. Rolandi E, Franceschini R, Cataldi A, Barreca T 1992 Endocrine effects of sumatriptan. Lancet 339: 13651368 621. Netti C, Guidobono F, Olgiati VR, Sibilia V, Pecile A 1981 Histamine agonist and antagonist drugs: interference with CNS control of GH release in rats. Horm Res 14: 180 191 Netti C, Guidobono F, Olgiati VR, Sibilia V, Pagani F, Pecile A 1982 Influence of brain histaminergic system on episodic growth hormone secretion in the rat. Neuroendocrinology 35: 43 47 Netti C, Guidobono F, Olgiati VR, Sibilia V, Pagani F, Pecile A 1984 Failure of somatostatin antiserum to reverse histamine-induced inhibition of pulsatile growth hormone secretion. Horm Res 19: 1217 624. Duggan DE, Hooke KF, Maycock AL 1984 Inhibition of histamine synthesis in vitro and in vivo by 5 fluoromethylhistidine. Biochem Pharmacol 33: 4003 4009 Grilli R, Sibilia V, Torsello A, Guidi M, Luoni M, Netti C, Mueller EE 1996 Role of neuronal histaminergic system in the regulation of somatotropic function: comparison between the neonatal and adult rat. J Endocrinol 151: 195201 and lactulose.
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