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Refuse to put stickers on my car because of one reason. I don't want to be shoved into a box and categorized as someone who puts stickers on their car. I don't want to be lumped in with the people who stand on soapboxes from behind their steering wheels. And I don't want people to think they know me without even saying hello. The clothes we wear, the products we use, the foods we eat, the bikes we ride, and the haircuts we have reveal some things about us. But finding out what's inside takes some effort. I can't tell that you're a nice person just because you wear a Red Sox cap. And I shouldn't immediately decide that you're a fanatic, granolacrunching freak if you don't chow down on foods without faces. I also shouldn't be astonished if you ride a 40-lb freeride steed and still blast past me on the uphills. You just can't tell what people are like from standing outside looking in. This entire label-wearing rant came to me in early July after I watched Lance Armstrong, Alexandre Vinokourov, Jan Ullrich and others crawl past me during stages 10 and 11 in the French Alps. As you probably know, this skinny-tire event is the marketing equivalent of the Republican Party in the United States. Le Tour de France is brilliant in how well it portrays itself that all of Europe is willing to spend 48 hours standing on the slopes of an Alp to see riders scream past at nearly 55 MPH on a downhill. Tour management is so well versed in what people want, that it can put together a team of nearly 30 cars in a pre-race 'Carnivale' every day, an hour before each stage of this 21-day race, and people will line up to catch virtually anything tossed at them. The Carnivale riders toss packets of cream cheese, lanyards with the word Cofidis on them, umbrella hats, and even stickers. And then these people-rabid with excitement for what they have just witnessed-will put these stickers on their cars, their clothing and their bodies. And then these people mostly greedy.
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17. Natelson BH, Cheu J, Pareja J, Ellis SP, Policastro T, Findley TW. Randomized, double-blind, controlled placebo-phase in trial of low dose of phenelzine in the chronic fatigue syndrome. Psychopharmacology 1996; 124: 226 McKenzie R, O'Fallon A, Dale J, et al. Low-dose hydrocortisone in chronic fatigue syndrome: a randomised controlled trial. J Med Assoc 1998; 280: 1061 Cleare AJ, Heap E, Malhi GS, Wessely S, O'Keane V, Miell J. Low-dose hydrocortisone in chronic fatigue syndrome: a randomised crossover trial. Lancet 1999; 353: 455 Rowe PC, Calkins H, DeBusk K, McKenzie R, Anand R, Sharma G, et al. Fludrocortisone acetate to treat neurally mediated hypotension in chronic fatigue syndrome: a randomized controlled trial. J Med Assoc 2001; 285: 52 Peterson PK, Pheley A, Schroeppel J, Schenck C, Marshall P, Kind A, et al. A preliminary placebo-controlled crossover trial of fludrocortisone for chronic fatigue syndrome. Arch Intern Med 1998; 158: 908 Forsyth LM, Preuss HG, MacDowell AL, Chiazze L Jr, Birkmayer GD, et al. Therapeutic effects of oral NADH on the symptoms of patients with chronic fatigue syndrome. Ann Allergy Asthma Immunol 1999; 82: 185 Natelson BH, Cheu J, Hill N, Bergen N, Korn L, Denny T, et al. Single-blind, placebo phase-in trial of two escalating doses of selegiline in the chronic fatigue syndrome. Neuropsychobiology 1998; 37: 150 Hickie I, Wilson A, Wright J. A randomised, double-blind placebo controlled trial of moclobemide in patients with chronic fatigue syndrome. J Clin Psychiatry 2000; 61: 643 Tiev KP, Cabane J, Imber JC. Treatment of chronic postinfectious fatigue: a randomized double-blind study of two doses of sulbutiamine 400 600 mg day ; versus placebo. Rev Med Interne 1999; 30: 912 Snorrason E, Geirsson A, Stefansoon K. Trial of a selective acetylcholinesterase inhibitor, galanthamine hydrobromide, in the treatment of chronic fatigue syndrome and related disorders. Presented at First World Congress on Chronic Fatigue Syndrome; Brussels, Belgium, 1995. 27. Moorkens G, Wynants H, Abs R. Effect of growth hormone treatment in chronic fatigue syndrome: a preliminary study. Growth Horm IGF Res 1998; 8: 131 Straus SE, Dale JK, Tobi M, Lawley T, Preble O, Blaese RM, et al. Acyclovir treatment of the chronic fatigue syndrome: lack of efficacy in a placebo-controlled trial. N Engl J Med 1988; 319: 1692 Vollmer-Conna U, Hickie I, Hadzi-Pavlovic D, Tymms K, Wakefield D, Dwyer J, et al. Intravenous immunoglobin is ineffective in the treatment of patients with chronic fatigue syndrome. J Med 1997; 103: 38 Peterson PK, Shephard J, Macres M, Schenk C, Crosson J, Rechtman D, et al. A controlled trial of intravenous immunoglobulin G in chronic fatigue syndrome. J Med 1990; 89: 554 Rowe KS. Double-blind randomized controlled trial to assess the efficacy of intravenous gamma globulin for the management of chronic fatigue syndrome in adolescents. J Psychiatr Res 1997; 31: 133.
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That is used for referring to a non-real world the world in which Mary did not leave ; . 3 . TIME: ONTOLOGY AND CONTENT In TOBI, the treatment of the linguistic phenomena illustrated in the previous section is based on the dichotomy between ontology and content. Informally speaking, ontology is the component of knowledge that has a general logical status; on the contrary, content is the component of knowledge that is highly situation dependent. As an example of this dichotomy, consider the case of subjective ; time. The ontology of time is its ordering and the fact that while the past is in a sense closed, the future is open. On the contrary, the metric of time is a content characteristic, in that the subjective evaluation of the duration of a time interval may vary depending on the situation. It is possible to split an inference process into two parts: an ontological part and a content one. In the former, only ontological inferences i.e., inferences based only on ontology, not on content ; will take place; in the latter, only content inferences will occur. In the same way, it is possible to speak of ontological knowledge and content knowledge. This division is rather approximate: there seems to be a deeper link between ontology and content than what is illustrated here; anyway it is interesting to study how far it is possible to push this dichotomy. I think that the phenomenon of temporal presuppositions can be explained in the following way: an event in the future cannot be certain, because of the partial unpredictability of the future. * This is why, using Gazdar's terminology, 'before' introduces a temporal presupposition, while 'after' does not. In implementing TOBI, I have assumed that ontology can be handled using classical symbolic methods; there are reasons, however, to believe that this might not be true for content see for example Airenti and Colombetti1 ; . Therefore, in the present version of TOBI, content inferences are replaced by an interface to an external user. This interface is activated upon request of a master module, which implements ontological inferences. 4 . RECURSIVE MODELS TOBI is a simple natural language comprehension system, able to understand a text sequence of utterances ; and to provide correct answers to questions regarding the text. I have considered only polar questions, i.e. questions admitting as answers only 'yes' true ; , 'no' false ; or 'I don't know' unknown ; . TOBI deals with text representation by building a model of the utterances, and it answers questions by evaluating them in the model. To understand how TOBI works, three notions must be examined: the notion of recursive model RM ; , the operation of building an RM from a text, and the operation of evaluating an utterance in an RM. To build a model of a text, a function that integrates a previous model with the information of a new utterance is needed; this function will be named int for 'integrate' ; . The function.
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International Chamber Music festival. MusicaNord is now offering, due to the high numbers of concerts it arranges each year, an international artist exchange program by interchanging concerts between norwegian and foreign artists.
Phocytic leukemia t-ANLL ; apy regimens that induce with ANLL de novo have t-ANLL.8" The higher and tolcapone.
E-00292-2004.R1 Page 13 underlying cartilage provides the best estimate of thyroidal SNS activity currently available. Responses of thyroid sympathetic nerves to various physiological manipulations are, in the main, consistent with known effects of these conditions on SNS activity in other tissues. Cold exposure is a well-known stimulus of SNS activity in many, but not all peripheral tissues 4, 8, 46, ; . The cold-induced acceleration in thyroidal NE turnover was qualitatively similar to that in heart Figure 3 ; and also consistent with indirect evidence of sympathetic activation in thyroid, since sympatholytic agents antagonize the cold-induced increase in colloid droplet formation in thyroid follicular cells in vivo 13 ; . The findings from the current experiment probably underestimate the impact of cold temperature since the controls were housed at 21C which is below the thermoneutral range for these animals 30 ; . Pregnancy also is associated with tissuespecific changes in SNS function 7 ; and its impact in thyroid was again qualitatively similar to that in heart Figure 4 ; . Consumption of a goitrogenic diet low iodine diet supplemented with PTU ; increased NE turnover in thyroid as well as heart. The stimulus for these responses likely represents a combination of both iodine deficiency and hypothyroidism, since hypothyroidism is widely recognized to stimulate sympathetic activity in human subjects 12, 29 ; and in peripheral tissues of experimental animals 2, 21, 36, ; . These studies also indicated that alterations in dietary intake affect thyroidal SNS activity. Fasting suppressed SNS activity in thyroid, a response similar to that shown previously in heart and other tissues 8, 43, 45, ; . While the role of local sympathetic innervation in the regulation of thyroid function is incompletely understood, it is likely that the reduction in thyroidal SNS activity Figure 4 ; contributes to the overall suppression in thyroid function during fasting 42 ; . On the other hand, diet may also.
Based technology can also be used for HPV DNA testing. Recently, Cytyc and Diogene, the manufacturer of the DNA-based Hybrid Capture II for detecting HPV, reached an agreement for Cytec to market DNA HPV testing in conjunction with ThinPrep Chea, January 19, 2001 ; . A combined screening test offers the opportunity for earlier detection and or longer screening intervals that could reduce cost. Studies are currently ongoing to evaluate the utility of ThinPrep specimens for detecting Chlamydia, which causes pelvic inflammatory disease PID ; . Women with Chlamydia often do not display outward symptoms, although it is a major cause of infertility among young women. Joint Pap and Chlamydia screening could be highly beneficial, particularly among young, sexually active women Hutchinson, personal communication and tolmetin.
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Tobi is on a quest for the incredible golden donuts, but nasty bullies are after him as well.
Evaluation in hypertransfused rats with selective radioiron probes of hepatocellular and reticuloendothelial iron stores and in iron-loaded rat heart cells in culture. Blood 97: 11151122 and topotecan.
In order to empirically study the relations between word informativeness and pitch accent, we use a medical corpus which includes a speech portion and a text portion. The speech corpus includes fourteen segments which total about 30 minutes of speech. The speech was collected at Columbia Presbyterian Medical Center CPMC ; where doctors informed residents or nurses about the postoperative status of a patient who has just undergone a bypass surgery. The speech corpus was transcribed orthographically by a medical professional and is also intonationally labeled with pitch accents by a ToBI Tone and Break Index ; Silverman et al., 1992; Beckman and Hirschberg, 1994 ; expert. The text corpus includes 1.24 million, 2, 422 discharge summaries, spanning a larger group of patients. The majority of the patients have also undergone cardiac surgery. The orthographic transcripts as well as the text corpus are used to calculate the IC and TF * IDF scores. First, all the words in the text corpus as well as the speech transcripts are processed by a stemming model so that words like "receive" and "receives" are treated as one word. We employ a revised version of Lovins' stemming algorithm Lovins, 1968 ; which is implemented in SMART. Although the usefulness of stemming is arguable, we choose to use stemming because we think.
Commutes since 2 kr T and by the above factorisation of 2 . Thus we can define : kr1 as the unique 2-cell such that p2 id and q2 1 . definition r and can be regarded as living in B T A, suffices to verify: rk 1, k id and r id. The key observation which enables us to perform these verifications is that since T is a parametric right 2-adjoint, the 2-cell T ; is a lax pullback in B T 1, because it is the result of applying the right 2-adjoint T1 to the lax pullback . To see that rk 1 first note that we have T ; rk T and toradol.
Has the patient had any prior systemic chemotherapy or pelvic irradiation? Is the patient receiving any potentially nephrotoxic or ototoxic drugs including aminoglycosides? Based on the urologist's, medical oncologist's and radiation therapist's opinions, is the patient medically stable to tolerate chemoradiation and cystectomy, if necessary? Has the patient undergone as thorough a transurethral resection of the bladder tumor as is judged safely possible? Does the patient have an adequately functioning bladder after evaluation by a urologist? Will treatment start within 6 weeks post TUR and endoscopic evaluations? Zubrod Performance status 1? Hemoglobin 10mg dl? WBC 4 ml per 1000 ; ? Platelet count 100 mm3 per 1000 ; ? ANC 1.8 mm3 per 1000 ; ? continued on page 2.
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Brewer M, Gershenson DM, Herzog CE, Mitchell MF, Silva EG and Wharton JT 1999 ; Outcome and reproductive function after chemotherapy for ovarian dysgerminoma. J Clin Oncol 17, 26702675. Cheong YC, Laird SM, Li TC, Shelton JB, Ledger WL and Cooke ID 2001 ; Peritoneal healing and adhesion formation reformation: review. Hum Reprod Update 7, 556566. Gershenson DM 1988 ; Menstrual and reproductive function after treatment with combination chemotherapy for malignant ovarian germ cell tumors. J Clin Oncol 6, 270275. Gershenson DM 1993 ; Update on malignant ovarian germ cell tumors. Cancer 71 Suppl 4 ; , 15811590. Gurgan T, Urman B, Yarali H and Duran HE 1997 ; Follicle-stimulating hormone levels on cycle day 3 to predict ovarian response in women undergoing controlled ovarian hyperstimulation for in vitro fertilization using a flare-up protocol. Fertil Steril 68, 483487. Kanazawa K, Suzuki T and Sakumoto K 2000 ; Treatment of malignant ovarian germ cell tumors with preservation of fertility: reproductive performance after persistent remission. J Clin Oncol 23, 244248. Kurman RJ 2002 ; Blaustein's Pathology of the Female Genital Tract, 5th edn. Springer--Verlag, New York. Low JJ, Perrin LC, Crandon AJ and Hacker NF 2000 ; Conservative surgery to preserve ovarian function in patients with malignant ovarian germ cell tumors. A review of 74 cases. Cancer 89, 391398. Mahdavi A, Pejovic T and Nezhat F 2006 ; Induction of ovulation and ovarian cancer: a critical review of the literature. Fertil Steril 85, 819826. Schwartz PE and Vidone RA 1981 ; Pregnancy following combination chemotherapy for a mixed germ cell tumor of the ovary. Gynecol Oncol 12, 373378. Tangir J, Zelterman D, Ma W and Schwartz PE 2003 ; Reproductive function after conservative surgery and chemotherapy for malignant germ cell tumors of the ovary. Obstet Gynecol 101, 251257. Tewari K, Rose GS, Balderston KD, Porto M and Kohler MF 1998 ; Fertility drugs and malignant germ-cell tumour of ovary in pregnancy. Lancet 351, 957958. Toth TL, Awwad JT, Veeck LL, Jones HW Jr, Muasher SJ 1996 ; . Suppression and flare regimens of gonadotropin-releasing hormone agonist. Use in women with different basal gonadotropin values in an in vitro fertilization program. J Reprod Med 41, 321326. Yoshinaka A, Fukasawa I, Sakamoto T, Tanaka M, Ota Y and Inaba N 2000 ; The fertility and pregnancy outcomes of the patients who underwent preservative operation followed by adjuvant chemotherapy for malignant ovarian tumors. Arch Gynecol Obstet 264, 124127. Zanagnolo V, Sartori E, Galleri G, Pasinetti B and Bianchi U 2004 ; Clinical review of 55 cases of malignant ovarian germ cell tumors. Eur J Gynaecol Oncol 25, 315320. Zanetta G, Bonazzi C, Cantu M, Binidagger S, Locatelli A, Bratina G and Mangioni C 2001 ; Survival and reproductive function after treatment of malignant germ cell ovarian tumors. J Clin Oncol 19, 10151020. Submitted on January 25, 2006; resubmitted on July 29, 2006, August 31, 2006; accepted on September 12, 2006 and toremifene.
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A study was made of 300 pregnancies occurring in 252 patients with organic cardiac disease who were treated in the Southern General Hospital, Glasgow. The incidence of cardiac disease was 3.6 per cent. The disease was rheumatic in origin in 92.3 per cent, and congenital in 6.6 per cent. When first examined, 35.3 per cent were in grade I, 57.3 per cent were in grade II, 6.3 per cent were in grade III, and 1 per cent were in grade IV. Early termination of pregnancy was only carried out in 3 patients 1 per cent ; in the present series. Pregnant cardiac patients should receive antenatal care at a special clinic at which both an obstetrician and a cardiologist see each patient together. All patients showing deterioration in the cardiac condition at any stage in pregnancy should be admitted at once, and every patient should be admitted at least 2 weeks before term. There is no place for cesarean section in the treatment of cardiac cases, and labor is generally reasonably rapid and easy. Sterilization, if considered advisable, is best carried out within a few hours of delivery under local anesthesia. There were 6 maternal deaths in 300 pregnancies 2 per cent ; . Of the remaining 294 pregnancies, the cardiac lesion appeared unaltered in 188. In 39 pregnancies, the cardiac condition was better on dismissal than when the patient was first seen. In 44 there was a temporary worsening and in only 23 was there any sustained cardiac deterioration. Four of the 6 deaths occurred in the early part of the series, and toward the end there were 126 consecutive deliveries without a maternal death. The uncorrected, combined fetal and neonatal mortality rate was 7.4 per cent and 6 babies at postmortem examination showed conditions incompatible with survival. BERNSTEIN.
Terazosin HCl.8 terbutaline sulfate .2 tetracycline HCl.4 Teveten HCT.18 Teveten.18 thiothixene.6 thiothixene HCl concentrate, oral.6 Tiazac.18 Tilade Inhaler.3 Timolide.18 timolol maleate.8 Tobi Ampul for Nebulization.5 Tofranil.17 tolazamide.10 tolbutamide .10 tolmetin sodium.14 Toprol XL.9 Tornalate .16 Tracer BG Test Strips .11 Tracleer.3 Trandate.18 Tranxene SD.17 Tranxene T-Tab.17 trazodone HCl .6 Tri-Levlen.19 Tri-Norinyl.19 triazolam.6 Tricor.9 trifluoperazine HCl.6 trimethoprim.4 Trinalin .16 Triphasil.13 U Uniretic .18 Univasc.18 V Vagifem.13 Valium.17 Vanceril.3 Vanceril DS .3 Vantin.17 Vaseretic.18 Vasotec.18 Velosef .17 Ventolin HFA.16 verapamil HCl.8 verapamil HCl capsule, 24 hr sustained release pellets .8 verapamil HCl tablet, sustained action.8 Vfend Suspension.17 Vfend Tablet.5 Vibramycin Suspension.5 Vibramycin Syrup .17 Vistaril .16 Vivactil .7 Vivelle Patch.13 Volmax.3 Voltaren.19 Vytorin.9 W Welchol.9 Wellbutrin SR.17 Wellbutrin XL .7 Wellbutrin.17 X Xanax XR .17 Xanax.17 Xopenex .3 and torsemide.
Factors in human carcinogenesis 8, 72 . The varied functions of P53 including control of the cell cycle, DNA repair and programmed cell death have earned it the name, "guardian of the genome". Mutant P53 protein has a half-life of approximately 24 hours compared to wild-type P53 protein which has a half-life of 20 minutes. This extended half-life of mutant P53 with lack of normal P53 ; allows it to accumulate and be over-expressed in tumors. The gene protein can then be easily recognized by immunohistochemical methods28 . Normal colonic epithelium is polyclonal having arisen from multiple stem cells . Studies of clonal composition of colonic tumors have revealed that all these tumors are monoclonal in composition . The morphological evidence of this clonal expansion has been studied by DNA ploidy analysis. Flowcytometry is a simple technique and allows rapid measurement of DNA content in a large number of cells. Several studies on the nuclear DNA content of colorectal tumors have suggested that DNA aneuploidy is an important marker for malianant transformation of these tumors and is related to poor prognosis 30-33 There has been increasing interest in tumor associated antigens in the colonic effluent for early diagnosis of colorectal cancer 34-38. Recently Tobi et al have described an Adenoma-associated-antigen defined by a monoclonal antibody designated Adnab-9 39. This 87-k Da protein antigen is present in a subpopulation of cells within an adenomatous polyp 40. The binding of antibody to this antigen in colonic effluent seems to correlate with the risk of colorectal cancer 40-41 Morphological and histological characters are the most important features of a polyp that correlate with its malignant potential. It is generally believed that the likelihood of invasive carcinoma increases with increased size of a polyp 42, 48 . Over 80% polyps found on colonoscopy are 1 cm in size and only 2-3% are larger than 3 cm. Only 1% of small 1 cm ; polyps have invasive cancer as compared to 10-20% of those larger than 2 cm 45. Large distal colonic polyps 1 cm ; are also associated with increased prevalence of synchronous proximal polyps with advanced pathology 46-47 . Several studies have demonstrated an increased risk of developing subsequent cancer in patients with larger than 1 cm polyps on initial examination 9, 47-48 Neoplastic polyps are histologically divided into tubular adenomas, villous adenomas and mixed or tubulo-villous adenomas. The National Polyp Study reported that 87% of 3358 adenomatous polyps were tubular, 5 % villous and 8% tubulo-villous 49 . The risk of malignant transformation is low in tubular adenomas 2-3% ; and high in pure villous adenomas 15-25% ; . The tubulo-villous adenomas have an intermediate risk of malignant transformation 45. Many studies have demonstrated that having multiple polyps at the time of initial diagnosis increases the risk of developing subsequent adenomas and adenocarcinomas A polyp is called pedunculated when it has a stalk. One of the definitions is that a polyp is pedunculated if the length of the stalk is greater than its diameter 51. In a review of literature, Coverlizza et al found that sessile polyps are more likely to have unfavorable histological features than pedunculated polyps 52. By definition all colorectal adenomas are dysplastic. The degree of dysplasia has been graded into mild, moderate and severe on the basis of cytological and architectural features. The degree of dysplasia has been associated with malignant transformation. About 90% of patients with carcinoma have high grade dysplasia in the resected specimens and some authors believe that 40% of resected polyps with high grade dysplasia have a focus of "carcinoma in situ"53 . To avoid confusion adenomas with severe dysplasia or "carcinoma in situ" have been called polyps with, "high grade dysplasia". It has been suggested that both "carcinoma in situ" and intramucosal carcinoma be reported as non-invasive carcinoma 54. The predominant opinion among gastrointestinal pathologists is that the spread of neoplastic cells through the muscularis mucosae should be considered to be an invasive carcinoma . A polyp with invasive carcinoma is also called a "malignant polyp". Complete endoscopic removal of an adenoma with a noninvasive carcinoma represents a cure. Although most of the polyps with invasive carcinoma are adequately treated by endoscopic polypectomy, about 10% patients will have residual cancer in the colonic wall or lymph nodes at the time of polypectomy or on follow-up 52, 54, 56 . Prognostic criteria have therefore been formulated to help in the proper management of these patients 52, 54, 56 . Prognostically favorable features include complete endoscopic resection, well differentiated histology, clear margin of resection and lack of vascular and or lymphatic invasion 52, 54, 56 RqfiqA. Sheikh, MBBS, MD, MRCP UK ; , FACP, FACG1; Shagnfta Yasmeen, MD, MRCOG London ; 2; Thomas Prindiville, MD3; B.H. Rnebnei; MD4. Department of Gastroenterology, San Joaquin General Hospital, Stockton, CA1; Department of Obstetrics Gynecology - ; Gastroenterology 3; Pathology4' University of California Davis Medical Center JK-Practitioner 2002; 9 4 ; : 215-218 REFERENCES and tobi.
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