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Example, thought that there was one such moment call it "the present, " if you like ; when the noumenal self constructs the entire causal sequence of all events in time. Most of us--followers of Walter Benjamin that we are--believe that there are more than one such moments. Certain moments in our lives, overcoded by collections of photographs, documents, and such paraphernalia, which mark onto-symbolic changes in the course of our lives--birth, marriage, graduation, certification, death, etc.--claim such a rich significance; though generally they do so fraudulently. The saturation of significance at these events is fraudulent because their possibility, if not their inevitability, is already explicable in advance. However, it is these conventional, explicable "saturated" moments which point the way to an uneasy comprehension of the "authentic" saturated moments from within the ideological realm of explicability in which we live. The distinction between "authentic" and "inauthentic" saturation uneasily drawn above will be illustrated herein by gesture to several recent films in which several scenarios of feminine transgression deviance are played through. This new distinction between transgression and mere deviance recodes that between authenticity and inauthenticity at a "higher" level; though the dis-ease we must feel at all of these is little assuaged by the variation in nomenclature. Words simply fail us. Nonetheless, let me try to sketch what I believe to be signified, however ephemerally, by this quadrangle of words: authentic, inauthentic.
Legislative or regulative action Country ISR NOR FRA Effective Date 1979 2 Feb 1979 Description of action taken Grounds for decision Withdrawn from the market following reports of increased mortality associated with use. Withdrawn from the market following reports of increased mortality associated with use. The indications have been restricted, as for every hypolipidaemic drug, to the treatment of endogenous hypercholesterolaemia and hypertriglyceridaemia a ; when a suitable and assiduously followed diet has proved inadequate; and b ; when cholesterolaemia is still raised after dieting and or there are associated risk factors present. Reference: FRAPC ; Press Communiqu , Feb 1979 ; Indications are restricted to treatment of patients with hyperlipidaemia refractory to dietary measures. Reference: FDADB ; FDA Drug Bulletin, 9 3 ; , 14, 1979 ; Severely restricted in use to certain patients only. This compound has been shown to cause hepatic tumours in rodents. There is an increased risk of malignancy and cholelithiasis with use in humans. A warning statement is required to be placed on the labels of all products. Currently marketed in Italy with limited therapeutic indications certain hyperproteinaemias with ascertained diagnoses; diabetic exudative retinopathy; xanthomes ; . Used only in cases of severe hyperlipoproteinaemia due to increased mortality connected with long-term treatment. Under the provisions of the Drugs Control ; Ordinance, this drug has been banned since it increases the incidence of gallstones and cholecystitis, drug-induced cardiac arrhythmias, cardiomegaly, angina, claudication and thromboembolic phenomena. It also enhances the effects and toxicity of other acidic drugs and it is implicated in the incidence of various tumours. Reference: BGDCO ; The Drugs Control ; Ordinance , 1982 ; Indications are restricted to treatment of patients with high plasma lipid levels, resistant to dietary control. Reference: CHLRS ; Resolution of the Minister of Health, 3261 Dec 1982 ; Indications are restricted to treatment of patients with hyperlipidaemia refractory to dietary measures. Reference: FDADB ; FDA Drug Bulletin, 9 3 ; , 14, 1979 ; Indications are restricted to treatment of patients with hyperlipidaemia. Indications are restricted to treatment of patients with hyperlipidaemia refractory to dietary measures. Reference: FDADB ; FDA Drug Bulletin, 9 3 ; , 14, 1979 ; Indications are restricted to treatment of patients with severe hyperlipidaemia. Currently available on the market. Precautionary information is required to be given with this drug. Severely restricted for use and or sale. Subject to restricted use and or sale. WHO Comment : Clofibrate, an antihyperlipidaemic agent, was introduced in 1967 and was subsequently extensively studied in the primary and secondary prevention of ischaemic heart disease. Following reports, published in 1978, of increased mortality among patients receiving clofibrate in a WHO-sponsored cooperative trial concerned with the primary prevention of ischaemic heart disease, the drug was withdrawn in some countries and its approved indications were severely restricted Legislative or regulation action.
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Your Company has achieved a turnover of Rs. 1605.20 millions registering a growth of 14.03 per cent over the previous year's turnover of Rs. 1407.68 millions. Profit before tax also increased from Rs.74.56 millions to Rs. 116.96 millions registering a growth of 56.87 per cent. A provision for deferred tax to the tune of Rs. 32.50 million was made during the financial year 2003-04. The exports of the Company also increased by 60.21 per cent from Rs. 423.99 million to Rs. 679.26 millions during the year. The earning per share increased to Rs. 5.24 from Rs. 4.78. The rise in margins was mainly due to higher exports and also due to: Focus on regulated markets. Steps taken to reduce the costs. Introduction on new products with higher margins.
Can be seen in Fig. 2, the animals put on a high-fat diet for 1 day exhibited a large and coordinated increase in LCAD 100% ; , MCAD 84% ; , and ACO 350% ; gene expression in the kidney cortex, compared with those kept on a low-fat diet. PPAR gene expression in 3-wk-old rat kidney: Effects of clofibrate on -oxidation enzyme gene expression. In Northern blots run from cortex and ISOM of 3-wk-old rats, the PPAR cDNA probe generated a single band of 8.5 kb, similar to that reported in studies of adult rat kidney or liver 22 ; . PPAR mRNA signals were fourfold higher in the cortex than in the ISOM Fig. 3 ; . Comparable amounts of PPAR transcripts were found in the immature cortex of 22-day-old rats fed low- or high-fat diet for 24 h Fig. 4 ; . Addition of 0.5% clofibrate to low-fat food for 1 day did not lead to significant change in PPAR mRNA, compared with the levels found in littermates on the low-fat diet only Fig. 4 ; . Switching pups onto a clofibrate-containing diet resulted in a marked and coordinated increase of LCAD 56% ; , MCAD 110% ; , and ACO 700% ; mRNA steady-state levels Fig. 5 ; . In contrast, no changes in MCAD gene expression occurred in response to clofibrate in the ISOM of the same animals data not shown ; . Short-term regulation of MCAD gene expression: Effects of lipids, clofibrate, and glucagon. To determine whether upregulation of -oxidation enzyme gene can and clorazepate.
He Boao Forum for Asia BFA ; will hold its Annual Conference 2006 from April 22 23, 2006 at its permanent home at the resort city of Boao in Hainan Province, China. The theme for the BFA Annual conference 2006 will be "Asia Searching for Win-win: Driving Growth to the Next Level". Highlights of this year's conference include sessions on trade and the "Doha Round", the international energy market, economic cooperation, real estate development with special sessions on China and Japan, and China and India. RPDEV Chairman Fidel V. Ramos, who is also the Chairman of the Board of.
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1. Berenson GS, Srinivasan SR, Bao WP 3rd, Tracy RE, Wattigney WA. Association between multiple cardiovascular risk factors and atherosclerosis in children and young adults. The Bogalusa Heart Study. N Engl J Med. 1998; 338: 1650-1656. The Third Report of the National Cholesterol Education Program NCEP ; Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults Adult Treatment Panel III ; . National Cholesterol Education Program. National Heart, Lung, and Blood Institute. National Institutes of Health. NIH Publication No. 02-5215, September 2002. 3. Grundy SM, Cleeman JI, Merz CN, Brewer HB Jr, Clark LT, Hunninghake DB, et al. Implications of Recent Clinical Trials for the National Cholesterol Education Program Adult Treatment Panel III guidelines. Circulation. 2004; 110: 227-239. Blankenhorn DH, Nessim SA, Johnson RL, Sanmarco ME, Azen SP, Cashin-Hemphill L. Beneficial effects of combined colestipol-niacin therapy on coronary atherosclerosis and coronary venous bypass grafts. JAMA. 1987; 257: 3233-3240. Erratum in: JAMA. 1988; 259: 2698. Brown G, Albers JJ, Fisher LD, Schaefer SM, Lin JT, Kaplan C, et al. Regression of coronary artery disease as a result of intensive lipid-lowering therapy in men with high levels of apolipoprotein B. N Engl J Med. 1990; 323: 1289-1298. Brown BG, Zhao XQ, Chait A, Fisher LD, Cheung MC, Morse JS, et al. Simvastatin and niacin, antioxidant vitamins or the combination for the prevention of coronary disease. N Engl J Med. 2001; 345: 1583-1592. Carlson LA, Rosenhamer G. Reduction of mortality in the Stockholm Ischaemic Heart Disease Secondary Prevention Study by combined treatment with clofibrate and nicotinic acid. Acta Med Scand. 1988; 223: 405-418 and clove.
JPET #49098 Fig. 8. Panel A: Western blot analysis of c-MYC, c-MYB, cyclin D1 D1 ; and cyclin D2 D2 ; protein levels in HL-60 cells treated with 50 M clofibrate and collected at the indicated times after the beginning of treatment. Equal protein loading was confirmed by exposure of the membranes to the anti-beta-actin antibody. Panel B: : Relative densitometric values of cMYC, c-MYB, cyclin D1 and cyclin D2 protein levels. Quantification of protein products was performed by densitometric scanning. Data are normalized using the beta-actin signal and are indicated as mean S.D. from three independent experiments and are expressed as percent of control value.
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Od1 to identify and isolate LDL subfractions among subjects with varying degrees of hypertriglyceridemia. Their physicochemical characteristics were compared with LDL subfractions isolated from the plasma of matched normolipidemic control subjects. Previously, we demonstrated that LDL subfractions in plasma from normolipidemic subjects differ in their tendency to undergo oxidative modification in vitro.7 In the present study, we investigated the susceptibility of the hypertriglyceridemic LDL subfractions to oxidation and related this to the content of antioxidants as well as the fatty acid pattern of LDL, with the goal of identifying a possible mechanism involved in the suggested increased atherogenicity of these lipoproteins.89 In addition, the influence of plasma triglyceride TG ; reduction by clofibrate treatment of the hypertriglyceridemic subjects, on both the physicochemical properties and the susceptibility to oxidation of the isolated LDL subfractions, was determined. Methods Subjects Twelve patients 11 men, one woman ; with various degrees of primary hypertriglyceridemia TG levels and cogentin.
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Tenderness as well as increases in creatine kinase and LD activities in serum. It is due to an as yet incompletely understood ; interference of clofibric acid derivatives with muscle cell metabolism. Interestingly, an increase in LD isoenzymes 1 and 2 in association with an "acute muscular syndrome" due to clofibrate has been mentioned in the literature 11 ; . We conclude that high proportions of LD isoenzyme 1 and a "flipped" LD 1 LD ratio may be present in rhabdomyolysis, as has been recently suggested by Nanji and Pudek 8 ; . Whether such an isoenzyme pattern might be common in the "acute muscular syndrome" secondary to clofibric acid derivatives remains to be established. References 1. Wright EJ, Cawley LP, Eberhardt L. Clinical application and interpretation of the serum lactic dehydrogenase zyrnogram. J Clin Pat wi 45, 737-745 1966.
Army policy restricted LSD tests to individual volunteers or small groups of military personnel. That was not enough for the leaders of the Chemical Corps. Major General Creasy bemoaned the fact that large-scale testing of psychochemical weapons in the United States was prohibited. "I was attempting to put on, with a good cover story, " he grumbled, "to test to see what would happen in subways, for example, when a cloud was laid down on a city. It was denied on reasons that always seemed a little absurd to me and cognex.
Over 9, 000 31% ; of the sampled members on cholesterol lowering medications have a confirmed history of heart disease, heart attack, stroke, or diabetes. Treatment to a LDL cholesterol level of 100 is important in this population.
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1. Holt S, Reeder B, Wilson M, Harvey S. Increased lipid peroxidation in patients with rhabdomyolysis: Lancet 1999; 353: 1241-3. Sekowski 1, Samuel P. Clofibrate - induced acute muscular syndrome. J Cardiol 1972; 30: 572-4. Pierides AM, Alvarez-Ude F, Kerr DNS, Skillen AW. Clofibrate induced muscle damage in patients with chronic renal failure. Lancet 1975; 2: 1279-82. Giraud P, Cassou M, Paul R, Guidet M. Toxicite musculaire due au fenofibriate: a propos d'un cas. Rev Rheum 1982; 49: 162. Haubenstock A, Schroecksnadel W, Bauer K, Schmidt P, Gabl F. Predominance of lactate dehydrogenase isoenzyme 1 in a patient with bezafibrate-induced rhabdomyolysis. Clin Chem 1984; 30: 1587-8. Yeshurun D, Dakak N, Khoury K, Daher E. Acute severe myositis due to bezafibrate treatment. Harefuah 1989; 116: 261-3. Gorriz JL, Sancho A, Lopez-Martin JM, Alcoy E, Catalan C, Pallardo LM. Rhabdomyolysis and acute renal failure associated with gemfibrozil therapy. Nephron 1996; 74: 437-8 and clofibrate
ABSTRACT Previous in vivo studies including those with knockout mice suggested that Niemann-Pick C1-like 1 NPC1L1 ; plays an essential role in the intestinal absorption of cholesterol. To characterize the mechanism of cholesterol uptake mediated by NPC1L1, an in vitro system reflecting the function of this transporter needs to be established. In the present study, we constructed NPC1L1 overexpressing CaCo-2 cells as an in vitro model and characterized the transport properties of NPC1L1. Immunohistochemical staining revealed that CaCo-2 cells express NPC1L1 on the apical membrane. It was also demon and colesevelam.
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8.1 EXTRAORDINARY GENERAL MEETING At the Company's Extraordinary General Meeting held 10 May 2001 shareholders approved all resolutions. Further details of these resolutions are contained in the Notice of Meeting and Explanatory Memorandum lodged with the ASX on 11 April 2001 and distributed to shareholders. The placements subsequent to shareholders' approval were made to Sons of Gwalia Limited 5.09 million ordinary shares ; and Imperial One Limited 6.45 million ordinary shares ; on 13 June 2001, bringing their respective interests to 19.9% and 24.36% as at the date of the Extraordinary General Meeting and raising approximately .8 million before costs ; . 8.2 OPTIONS During the June 2001 quarter, 47, 281 September 2001 options for fully paid ordinary shares were exercised at 20 cents each. Total exercise funds received totaled , 456.20.
Appears to be an important clinical variable that also identifies patients with a higher risk of sudden cardiac death.50 Arrhythmogenic right ventricular dysplasia is a particular kind of cardiomyopathy responsible for sudden death in young individuals and adults, with a gene defect recently localized to chromosomes 1 and 14 q23-q24.11, 52 It occurs as a familial disorder in 30% of cases, with autosomal dominant inheritance. Exercise can precipitate ventricular tachycardia in these patients, with an annual incidence of sudden death estimated to be 2%. Two pathological patterns, fatty and fibrofatty myocardial infiltration, have been identified. In the fibrofatty variety, myocardial atrophy appears to be the consequence of acquired injury and myocyte death and repair by fibrofatty replacement, mediated by patchy myocarditis. Apoptosis may be important. The left ventricle and ventricular septum can be involved in 50% to 67% of cases, often later in the disease, confirming a poor prognosis.52 ECG during sinus rhythm often exhibits T-wave inversion in V1 to complete or incomplete right bundle-branch block, and the ventricular tachycardia has a left bundle-branch block contour, with the frontal-plane axis reflecting the site of origin in 1 of predilection sites for ventricular fatty degeneration: right ventricular inflow and outflow tracts and apex, the so-called "triangle of dysplasia." During sinus rhythm, intraventricular conduction may be sufficiently slow as to produce a terminal notch on the QRS complex that Fontaine called an epsilon wave Figure 6 and colestipol.
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